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99.5% Adenosine Monophosphate Weight Loss , CAS 61-19-8 AMP Adenosine Phosphate

99.5% Adenosine Monophosphate Weight Loss , CAS 61-19-8 AMP Adenosine Phosphate

  • 99.5% Adenosine Monophosphate Weight Loss , CAS 61-19-8 AMP Adenosine Phosphate
  • 99.5% Adenosine Monophosphate Weight Loss , CAS 61-19-8 AMP Adenosine Phosphate
  • 99.5% Adenosine Monophosphate Weight Loss , CAS 61-19-8 AMP Adenosine Phosphate
  • 99.5% Adenosine Monophosphate Weight Loss , CAS 61-19-8 AMP Adenosine Phosphate
99.5% Adenosine Monophosphate Weight Loss , CAS 61-19-8 AMP Adenosine Phosphate
Product Details:
Place of Origin: Hubei,China
Brand Name: Bodybiological
Certification: ISO9001, SGS
Model Number: 61-19-8
Payment & Shipping Terms:
Minimum Order Quantity: 10gram
Price: USD1/gram
Packaging Details: Foil bag
Delivery Time: within 24 hours after the payment
Payment Terms: T/T, Western Union, MoneyGram
Supply Ability: 100kg/month
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Detailed Product Description
Keywords: Adenosine Monophosphate Alias: AMP
CAS: 61-19-8 Appearance: White Powder
Purity: 99.5% Shipping Method: Fedex, TNT, DHL, UPS, HK EMS
Payment Terms: Money Gram, Western Union, Bank Transfer Export Market: USA, UK, Brazil, Thailand, France, Canada
Policy: Reshipping Policy Brand: Bodybiological
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99.5% Antiasthmatic Adenosine Monophosphate AMP Pharmaceuticals Raw Materials CAS 61-19-8

 

whatsapp:+8613429837396

 

Basic information:

 

Product name:

Adenosine 5'-monophosphate

Other Name

a5mp;adenosine,mono(dihydrogenphosphate)(ester);adenosine5'-phosphate;adenovite;adenyl;amp(nucleotide);amp[nucleotide];cardiomone

Original

China

CAS

61-19-8

Molecular formula

C10H14N5O7P

Brand name

NJBNSteroid

Delivery time

within 24 hours upon receipt of payment

Delivery

Fast and secure shipping by EMS, DHL, TNT, FedEx, UPS

Purity

99%

Package

Discreet and Hidden package according to specific requirement

Appearance

White Powder

Certification

SGS, ISO 9001, KOSHER

Payment Terms

Western Union, Money Gram , T/T, Bitcoin

Minimum Order Quantity

10 g

Price

Negotiable

Usage

antiasthmatic

 

Detailed information:

 

Adenosine monophosphate (AMP), also known as 5'-adenylic acid, is a nucleotide that is used as a monomer in RNA. It is an ester of phosphoric acid and the nucleoside adenosine, which does not have the typical high energy bond of the so called adenosine monophosphate ("AMP") that arises on the hydrolysis of ATP. AMP consists of a phosphate group, the sugar ribose, and the nucleobase adenine. As a substituent it takes the form of the prefix adenylyl-.

 

Adenosine monophosphate (AMP) deaminase deficiency is a condition that can affect the muscles used for movement (skeletal muscles). In many affected individuals, AMP deaminase deficiency does not cause any symptoms. People who do experience symptoms typically have fatigue, muscle pain (myalgia), or cramps after exercise or prolonged physical activity (exercise intolerance). Following strenuous activity, they often get tired more quickly and stay tired longer than would normally be expected. In rare cases, affected individuals have more severe symptoms including severe muscle weakness, low muscle tone (hypotonia), and muscle wasting (atrophy), but it is unclear whether these symptoms are due solely to AMP deaminase deficiency or additional health conditions. Exercise intolerance associated with AMP deaminase deficiency usually becomes apparent in childhood or early adulthood.

 

Application:

 

1. Triethanolamine buffer, 0.1M, pH 7.6: 1.86 g TEA• HCl in 80 ml distilled water. Adjust to pH 7.6 with 1.0 M NaOH, adjust volume to 100 ml with distilled water.
2. 14mM NADH:10 mg NADH-Na2 with 1 ml distilled water.
3. 16.5 mM ATP: 10 mg NADH-Na2 in 1 ml distilled water.
4. 0.5 M MgSO4/2 M KCl: 1.23 g MgSO4• 7 H2O and 1.49 g KCl in 10 ml distilled water.
5. 32 mM Phosphoenolpyruvate: 15 mg PEP-(CHA)3 in 1 ml MgSO4/KCl.
6. LDH, from rabbit muscle: 5 mg protein/ml (550 U/mg).
7. Pyruvate kinase, from rabbit muscle: 10 mg protein/ml (200 U/mg).
8. Myokinase, from rabbit muscle: 5 mg protein/ml (360 U/mg).

 

Function:

 

AMP is a second messenger, used for intracellular signal transduction, such as transferring into cells the effects of hormones like glucagon and adrenaline, which cannot pass through the plasma membrane. It is also involved in the activation of protein kinases. In addition, cAMP binds to and regulates the function of ion channels such as the HCN channels and a few other cyclic nucleotide-binding proteins such as Epac1 and RAPGEF2

 

cAMP and its associated kinases function in several biochemical processes, including the regulation of glycogen, sugar, and lipid metabolism.
In eukaryotes, cyclic AMP works by activating protein kinase A (PKA, or cAMP-dependent protein kinase). PKA is normally inactive as a tetrameric holoenzyme, consisting of two catalytic and two regulatory units (C2R2), with the regulatory units blocking the catalytic centers of the catalytic units.
Cyclic AMP binds to specific locations on the regulatory units of the protein kinase, and causes dissociation between the regulatory and catalytic subunits, thus enabling those catalytic units to phosphorylate substrate proteins.


The active subunits catalyze the transfer of phosphate from ATP to specific serine or threonine residues of protein substrates. The phosphorylated proteins may act directly on the cell's ion channels, or may become activated or inhibited enzymes. Protein kinase A can also phosphorylate specific proteins that bind to promoter regions of DNA, causing increases in transcription. Not all protein kinases respond to cAMP. Several classes of protein kinases, including protein kinase C, are not cAMP-dependent.
Further effects mainly depend on cAMP-dependent protein kinase, which vary based on the type of cell.
Still, there are some minor PKA-independent functions of cAMP, e.g., activation of calcium channels, providing a minor pathway by which growth hormone-releasing hormone causes a release of growth hormone.
However, the view that the majority of the effects of cAMP are controlled by PKA is an outdated one. In 1998 a family of cAMP-sensitive proteins with guanine nucleotide exchange factor (GEF) activity was discovered. These are termed Exchange proteins activated by cAMP (Epac) and the family comprises Epac1 and Epac2.[citation needed] The mechanism of activation is similar to that of PKA: the GEF domain is usually masked by the N-terminal region containing the cAMP binding domain. When cAMP binds, the domain dissociates and exposes the now-active GEF domain, allowing Epac to activate small Ras-like GTPase proteins, such as Rap1.

 

Dosage:

 

The recommended Adenosine injection dose is 0.14 mg/kg/min infused over six minutes (total dose of 0.84 mg/kg) (Table 1).

Administer Adenosine injection only as a continuous peripheral intravenous infusion

Inject Thallium-201 at the midpoint of the Adenosine injection infusion (i.e., after the first three minutes of Adenosine injection)

Thallium-201 is physically compatible with Adenosine injection and may be injected directly into the Adenosine injection infusion set

Inject Thallium-201 as close to the venous access as possible to prevent an inadvertent increase in the dose of Adenosine injection (the contents of the intravenous tubing) being administered
Visually inspect Adenosine injection for particulate matter and discoloration prior to administration. Do not administer Adenosine injection if it contains particulate matter or is discolored.
There are no data on the safety or efficacy of alternative Adenosine injection infusion protocols. The safety and efficacy of Adenosine injection administered by the intracoronary route have not been established.

 

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99.5% Adenosine Monophosphate Weight Loss , CAS 61-19-8 AMP Adenosine Phosphate 0 

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